Please use this identifier to cite or link to this item: http://repository.tma.uz/xmlui/handle/1/1019
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dc.contributor.authorSH.A.Saidova, A.V. YAkubov, D.B.Pulatova, N.I.Pulatova-
dc.date.accessioned2021-11-02T09:55:38Z-
dc.date.available2021-11-02T09:55:38Z-
dc.date.issued2021-
dc.identifier.urihttp://repository.tma.uz/xmlui/handle/1/1019-
dc.description.abstractStenokardiya negizida kislorodni etkazib berish va miokardning kislorodga bo'lgan ehtiyoji orasida nomutanosiblik yotadi. Kislorodni etkazib berishning pastligi epikardial qon tomirlarning aterosklerozi oqibatida, yoki vazokonstriksiya sababli yuzaga kelgan to'siq natijasi, yoki epikardial koronar arteriyalarning yoki kichik diametrdagi intramiokardial arteriyalarning kengayish hossasi yo'qligi oqibati hisoblanishi mumkin. Nostabil stenokardiya yoki MI mavjud bemorlarda aterosklerotik pilakchaning uzilishidan yuzaga kelgan ikkilamchi tromboz koronar qon tomirlardagi to'siq bo'lishi mumkin. Koronar arteriyalardagi aterosklerotik jarayon endoteliy disfunksiyasi bilan birga kechadi, endotelial disfunksiya tomirlarni kengaytiruvchi kuchli qon aylanishi, jismoniy yuklama, taxikardiya, atsetilxolin va sovuqli sinama kabi oddiy qo'zg'atish omillaiiga javob tariqasidagi koronar tomir to'rining dilatatsiya qila olmaslik ko'rinishida namoyon bo'lishi mumkin.en_US
dc.language.isootheren_US
dc.publisherToshkenten_US
dc.subjectYurak ishemik kasalligi, dori vositalarini ratsional qo'llash, klinik farmakologik yondashuven_US
dc.titleYurak ishemik kasalligini davolashda dori vositalarini ratsional qo'llashga klinik farmakologik yondashuven_US
dc.typeArticleen_US
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