Please use this identifier to cite or link to this item: http://repository.tma.uz/xmlui/handle/1/9119
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dc.contributor.authorEshmamatov O.F., Turakulov R.I.-
dc.date.accessioned2023-10-03T18:32:39Z-
dc.date.available2023-10-03T18:32:39Z-
dc.date.issued2023-
dc.identifier.urihttp://repository.tma.uz/xmlui/handle/1/9119-
dc.description.abstractYaqin vaqtgacha buyrak qon oqimining buzilishi, ba’zi dori-darmonlarni qabul qilish va chekish tufayli eritropoetin sintezining kuchayishi bilan bog‘liq kompensator eritrositoz o‘pkaning surunkali obstruktiv kasalligi (O‘SOK) ning uzoq muddatli kechishining asosiy belgilaridan biri hisoblangan. Yaqinda o‘tkazilgan bir qator tadqiqotlar O‘SOK bilan og‘rigan bemorlarda normoxrom normositar anemiyani ko‘rsatdi. O‘SOKda kamqonlikni uchrashini o‘rganish natijalari juda katta farq qildi - 7,5 dan 33% gacha. Kamqonlik O‘SOKning tizimli namoyon bo‘lishimi yoki qo‘shma kasallikmi degan savol hozirda dolzarb masala. Maqolada O‘SOK va kamqonlik o‘rtasidagi bog‘liqlik muammosi bo‘yicha adabiyotlarni ko‘rib chiqish keltirilgan.en_US
dc.publisherВестник ТМАen_US
dc.subjecto‘pkaning surunkali obstruktiv kasalligi, anemiya, eritrositoz, normoxrom, eritropoez.en_US
dc.titleO‘PKANING SURUNKALI OBSTRUKTIV KASALLIGIDA KAMQONLIKNI UCHRASHIen_US
dc.typeArticleen_US
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