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DC Field | Value | Language |
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dc.contributor.author | Madina Bohodirovna Rakhimova, Khalmurad Sadullaevich Akhmedov, Sagdiyana Nasurullaevna Buranova | - |
dc.date.accessioned | 2022-10-05T14:58:26Z | - |
dc.date.available | 2022-10-05T14:58:26Z | - |
dc.date.issued | 2022 | - |
dc.identifier.citation | Тошкент | en_US |
dc.identifier.issn | 2181-5534 | - |
dc.identifier.uri | http://repository.tma.uz/xmlui/handle/1/2442 | - |
dc.description.abstract | Despite the fact that the attention of a huge number of researchers around the world is focused on the development of effective clinical protocols and recommendations for the management of patients with a new coronavirus infection, the presence of a large number of severe forms of the disease and deaths allows usto count, that to date, doctors have not formed a holistic picture of the pathogenesis of this disease, enabling effective atment and prevention of critical complications. In a number of studies in patients with severe COVID-19, a statistically significant association was found between the detection of icrotrombs in the pulmonary vessels, disorders of the coagulation balance and damage to the vascular endothelium [1, 2, 3]. However, when describing the pathogenesis of COVID-19, as well as the factors involved in the development of complications of a new coronavirus infection, insufficient attention is paid to endothelial dysfunction. | en_US |
dc.language.iso | other | en_US |
dc.relation.ispartofseries | УДK;616.721-002.77-085.814.1: 578.834.1 | - |
dc.subject | Effect of the CD147 marker (basigin, EMMPRIN, transmembrane serine protease) on the development of endothelial damage in COVID-19 | en_US |
dc.title | Endothelial dysfunction as a link in COVID-19 pathogenesis | en_US |
dc.type | Article | en_US |
Appears in Collections: | Thesis, Articles |
Files in This Item:
File | Description | Size | Format | |
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2 Madina Bohodirovna Rakhimova, Khalmurad Sadullaevich Akhmedov,.pdf | Endothelial dysfunction as a link in COVID-19 pathogenesis | 370.13 kB | Adobe PDF | View/Open |
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