Please use this identifier to cite or link to this item: http://repository.tma.uz/xmlui/handle/1/5336
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dc.contributor.authorYuan Xu, Mahmood Al-Mualm, Ermias Mergia Terefe, Maksuda Ilyasovna Shamsutdinova, Maria Jade Catalan Opulencia, Fahad Alsaikhan, Abduladheem Turki Jalil, Ali Thaeer Hammid, Ayesheh Enayati, Hassan Mirzaei i, Vahid Khori i , Ali Jabbari i , Aref Salehi i, Alireza Soltani, Abdullah Mohamed-
dc.date.accessioned2022-12-16T06:49:02Z-
dc.date.available2022-12-16T06:49:02Z-
dc.date.issued2022-
dc.identifier.urihttp://repository.tma.uz/xmlui/handle/1/5336-
dc.description.abstractIn the novel SARS-CoV-2 (COVID-19) as a global emergency event, the main reason of the cardiac injury from COVID-19 is angiotensin-converting enzyme 2 (ACE2) targeting in SARS- CoV-2 infection. The inhibition of ACE2 induces an increase in the angiotensin II (Ang II) and the angiotensin II receptor type 1 (AT1R) leading to impaired cardiac function or cardiac inflammatoryen_US
dc.language.isoen_USen_US
dc.publisherArabian Journal of Chemistryen_US
dc.relation.ispartofseriesУДК;-
dc.subjectCOVID-19; Potentilla reptans; Angiotensin II;en_US
dc.titlePrediction of COVID-19 manipulation by selective ACE inhibitory compounds of Potentilla reptant root: In silico study and ADMET profileen_US
dc.typeArticleen_US
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