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DC Field | Value | Language |
---|---|---|
dc.contributor.author | Axmedov X., Raximov S. | - |
dc.date.accessioned | 2023-11-26T15:44:10Z | - |
dc.date.available | 2023-11-26T15:44:10Z | - |
dc.date.issued | 2023 | - |
dc.identifier.citation | Toshkent | en_US |
dc.identifier.issn | 2181-7812 | - |
dc.identifier.uri | http://repository.tma.uz/xmlui/handle/1/9420 | - |
dc.description.abstract | Ankilozlovchi spondiloartrit (AS) patogenezida ishtirok etadigan sitokin IL-17 darajasi kasallikning faolligi bilan bog‘liq va u anti-TNF-a ga javob bermaydigan AS bemorlarini davolashda asos sifatida ishlatiladi. Ushbu tadqiqotda IL-17 ning ankilozlovchi spondiloartrit kasalligi faollik ko‘rsatkichi C-reaktiv oqsil (ASDAS-CRO) bilan o‘lchanadigan kasallik faolligi bilan bog‘liqligi o‘rganildi. Tadqiqotda korrelyatsiya kuchini ko‘rish uchun Spearman’s korrelyatsiya testi yordamida kasallik faolligi va IL-17 darajasi tahlil qilindi. Qirqta AS ga chalingan bemorlarning o‘rtacha Yoshi 53,58 ± 9,28 yil, tana vazni indeksi 24,36 ± 3,23 kg / m2, ECHT 39,50 ± 18,76 mm / soat, klinik olingan Shober sinamasi13,11 ± 1,27 sm, tranzit testi ± 1,27 sm. O‘rtacha CRO va IL-17 mos ravishda 0,3 (0,10-5,70) mg / dL va 9,30 (7,70- 13,60) pg / dL ni tashkil etdi . ASDAS-CRO tizimiga asoslanib, bemorlar yuqori (62,5%), o‘rtacha (35%) va harakatsizlik (2,5%) toifasiga kiruvchi kasallik faolligini ko‘rsatdi. IL-17 darajasi AS bilan kasallangan bemorlarda kasallik faolligi bilan kuchli bog‘liqdir. | en_US |
dc.language.iso | other | en_US |
dc.relation.ispartofseries | УДК;616.711-002.72-007.274 -085 | - |
dc.subject | Ankilozlovchi spondiloartrit; IL-17; ASDAS; BASDAI; Shober sinamasi | en_US |
dc.title | Ankilozlovchi spondiloartritda IL-17 ning kasallik kechishidagi axamiyati | en_US |
dc.type | Article | en_US |
Appears in Collections: | Thesis, Articles |
Files in This Item:
File | Description | Size | Format | |
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46. ВЕСТНИК Axmedov X.S Raximov S.S | Ankilozlovchi spondiloartritda IL-17 ning kasallik kechishidagi axamiyati | 696.4 kB | Unknown | View/Open |
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