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«Scientific substantiation of histological changes in the pulmonary endothelium in diabetes».

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dc.contributor.author 1. Sobirova D.R.
dc.contributor.author Usmanov R.J., Azizova F.X.
dc.date.accessioned 2021-12-27T08:53:33Z
dc.date.available 2021-12-27T08:53:33Z
dc.date.issued 2021-10-10
dc.identifier.issn 2776-0979
dc.identifier.uri http://repository.tma.uz/xmlui/handle/1/1117
dc.description.abstract Diabetes mellitus is a disease of the endocrine system caused by a complete or partial deficiency of the hormone insulin. As a result, hyperglycemia can be observed - a continuous increase in blood glucose levels. The disease is characterized by chronic course and impaired metabolic processes in the body (metabolism of carbohydrates, fats, proteins, minerals and water). Today, the prevalence of this disease in the population is 2%, of which 5-8% are children under 15 years. Evidence shows that the number of patients with diabetes is growing worldwide. The fate of patients with diabetes has so far been very sad, as they died in a short time (weeks or months). After the release of insulin, such patients were able to live longer. The risk of the disease can be passed from generation to generation in a dominant or recessive way. If the disease is dominant, the "risk group", the characteristics of the unfavorable, negative factors prevail. b Viral infections that can damage cells, long term eating disorders (high intake of carbohydrates and fats), various mental and physical injuries, obesity, complicated pregnancy, endocrine diseases (diffuse toxic goiter, acromegaly), caused by some somatic diseases - pancreatitis, hypertension. A non-genetically related type of diabetes mellitus - pancreatectomy - can be associated with pancreatic cancer en_US
dc.language.iso other en_US
dc.subject diabetes, disease prevalence, endocrine diseases, insulin, endothelial dysfunction of the pulmonary artery, pulmonary tuberculosis. en_US
dc.title «Scientific substantiation of histological changes in the pulmonary endothelium in diabetes». en_US
dc.type Article en_US


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